IndraLab

Statements

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"In addition, upregulation of Kcnk2 or knockdown of Foxd3 promoted the cell viability and reduced the apoptosis of the H/R-treated PC12 cells."
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"TREK-1 deficiency promoted apoptosis in HO exposed lungs."
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"However, the overexpression of Kcnk2 or knockdown of Foxd3 enhanced the cell viability and inhibited the apoptosis of the hypoxia/reoxygenation-treated PC12 cells (56)."
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"Furthermore, in an in vivo model of Acute Lung Injury (ALI) we recently found that TREK-1 deficiency led to increased lung damage and AEC apoptosis but decreased BAL cytokine levels [XREF_BIBR]."
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