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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

Beta-adrenoceptor activates KCNMA1. 2 / 2

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"These results suggest that beta-adrenoceptor stimulation increases maxi-K channel activity via a membrane delimited pathway, probably through pertussis toxin sensitive G protein (Gi)."

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"We conclude that the BK channel in cerebrovascular smooth muscle cells can be activated by beta-adrenoceptor stimulation, that the effect depends strongly on [Ca2+] i, and that the effect is mediated by cAMP dependent protein kinase A with no important contribution from a direct G protein or phosphorylation independent mechanism."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...