IndraLab

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Cocaine activates SCN1A. 2 / 2
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"These data suggest that the induction of NAC-1 by cocaine may be a compensatory mechanism that minimizes the behavioral impact of cocaine administration by regulating postsynaptic dopamine transmission within the nucleus accumbens."

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"Another factor, NAC-1, which is upregulated by cocaine, tends to oppose behavioral sensitization caused by cocaine ( Wang et al., 2003 )."