IndraLab

Statements

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"The dietary supplementation of selenium in mice produced anti-inflammatory activity by inhibiting the activation of the NALP3 inflammasome and nuclear factor-kappaB and mitogen activated protein kinase (NF-kappaB and MAPK) pathway in S. aureus induced mastitis."
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"Intriguingly, Se supplementation could suppress the AgNP exposure-induced NLRP3 inflammasome and inhibited the increase of ROS in AgNP-exposed rats and HUVECs, this was consistent with findings from previous studies [43, 44]."
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"Also, a study has reported that dietary selenium attenuates Staphylococcus aureus mastitis in mice by inhibiting the NLRP3 inflammasome ."
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"Selenium can reduce inflammatory damage by inhibiting the activation of the NLRP3 inflammasome pathway (Huang et al., 2021; Ma et al., 2019)."
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"In this study, we explored whether selenium can inhibit the activation of the NALP3 inflammasome and NF-kappaB and MAPK pathway to achieve anti-inflammatory effects."
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"which indicated that Se deficiency might cause an increase in NLRP3 inflammasomes and triggered pyroptosis ( Fig. 2 A)."
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"In vitro and animal studies have demonstrated that selenium inhibits the activation of the NLRP3 inflammasome, a key driver of inflammation, thereby reducing the production of pro-inflammatory cytokines such as IL-1β and IL-18 [69,70,71]."
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"The results revealed that the transcript (Figures 5A and 5B) and protein (Figures 5H–5I) levels of NLRP3 and ASC in the BPA group or Se- group were obviously higher than those in the C group, and the expression was further elevated in the BPA+Se- group (P < 0.05), showing that Se deficiency and BPA exposure activate NLRP3 inflammasome in the muscular stomach."
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"Se deficiency can lead to an increase in ROS and mediate the activation of NLRP3 inflammasomes, leading to Staphylococcus aureus-induced inflammation in bovine mammary epithelial cells [11] ."
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"A selenium-modified phytosomal tripterine was found to reduce cytotoxicity and inflammation by inhibiting the NLRP3 inflammasome and pyroptosis [201]."
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"Yang et al. [ 8 ] found that oral administration of selenium nanoparticles obviously inhibited the activation of NLRP3 inflammasome and reduced Aβ aggregation through reshaping the gut microbiota in A[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Se also can inhibit the activation of NLRP3 inflammasomes in broiler so as to relieve the inflammatory damage of broiler kidneys caused by lead [14] ."
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"Se nanoparticles may alleviate AKI induced by ischemia reperfusion injury by upregulated the (GPx)-1 levels and suppressed NLRP3 inflammasome [32]."
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"Se supplementation also inactivated the Sting pathway (stimulator of interferon genes) [65], which could promote transcription of interferon regulatory factor 3 (IRF3), activate NF-κB and the NLRP3 inflammasome, and induce ER stress, inflammation and apoptosis [66]."
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"In this experiment, we investigated whether selenium can inhibit the activation of the NLRP3 inflammasome in a mouse model of Staphylococcus aureus induced mastitis."
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"While further investigation is needed to elucidate the relationship between FAT3/NLRP3 and selenium metabolism, there is some evidence that selenium may inhibit the NLRP3 signaling pathway ."
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"Se Treatment Inhibited the Activation of the NLRP3 Inflammasome in AgNP-Administered Rats and HUVECs."
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