IndraLab

Statements

SCN4A binds HTT. 2 / 2
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"To evaluate if the genetic interaction between the mutated forms of huntingtin and Scn4a could enhance HD pathology in the brain, we measured soluble mutant huntingtin and huntingtin intranuclear inclusions at piriform cortex and cerebellum ( xref , Fig. S2D and E), finding no differences between genotypes."
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"The interaction of Scn4a and HD mutations leads to higher energy demands that trigger adaptation changes in skeletal muscles."
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