IndraLab

Statements

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"Accumulating evidence has demonstrated that USP37 promotes lung cancer cell proliferation, migration, and invasion XREF_BIBR but inhibits lung cancer cell apoptosis in a deubiquitination dependent manner."
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"USP37 up-regulation promoted the proliferation, cell cycle progression, apoptosis inhibition, migration, invasion, epithelial mesenchymal transition (EMT) and stemness of CRC cells; moreover, USP37 facilitated the angiogenesis of human umbilical vein endothelial cells (HUVECs)."
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"The results showed that USP37 knockdown significantly enhanced Sorafenib-triggered apoptosis in Huh7, SMMC-7721 and HepG2 cell lines (Fig. 4a and Figure S3 b,c)."
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"Depleting USP37 in osteosarcoma cells results in reduced recruitment to the replication fork, leading to instability of binding proteins, stalled replication fork, and enhanced apoptosis (Fig. 10B)."
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"USP37 knockdown repressed viability, EdU positive cell rate, promoted apoptosis rate in keloid fibroblasts, but these effects were reverted by SALL4 upregulation (Fig. 5B-D)."
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"The combined approach of USP37 knockdown and Dox treatment significantly increases cleaved Caspase 3 and Bax levels while suppressing BCL2 expression, resulting in cell cycle arrest and enhanced apoptosis [86]."
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