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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP15 activates immune response. 4 / 4

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"Among these proteins, we found Ubiquitin carboxyl-terminal hydrolase 15 (Usp15) which positively regulates type I interferon responses and thereby antiviral immune response."

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"Our previous work demonstrates that USP15 deficiency promotes antitumor immunity in a transplantable B16 melanoma tumor model, which differs from the present findings obtained using the MCA induced primary tumor model."

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"These findings indicate that USP15 ablation promotes immunity against transplantable MCA-205, as well as B16, tumors."

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"USP15 deficiency promotes immunity against transplanted MCA tumors."

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USP15 activates immune response. 4 / 4

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