IndraLab

Statements

USP14 activates triglyceride. 6 / 6
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"USP14 can promote hepatic triglyceride accumulation by increasing the stability of fatty acid synthase (FASN), which is the crucial enzyme for hepatic adipogenesis (150)."
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"To determine whether the increase of TG retention by USP14 overexpression relies on its regulation of FASN, we carried out adenoviral shRNA mediated FASN knockdown combined with USP14 overexpression in C57BL/6 mice."
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"For example, USP14 overexpression stimulates liver triglyceride accumulation by stabilizing fatty acid synthase [38]."
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"It is currently known that USP14 promotes hepatic triglyceride accumulation and produces steatosis by stabilizing FASN [ 15 ]."
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"For example, USP14 overexpression could promote the accumulation of liver triglyceride accumulation via stabilization of fatty acid synthase (FASN) in obese mice, resulting in hepatosteatosis, insulin resistance and hyperglycemia [9]."
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"These data emphasized that TGT stimulated TG catabolism, leading TG returned to normal level, by up-regulating the mRNA expression of ATGL rather than HSL in the kidney of mice with DN.TGT has a long [MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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