IndraLab

Statements

SCN5A binds RBM25. 6 / 6
| 4 2
sparser
"In summary, AngII and hypoxia, signals common to HF, result in increased hLuc7A and RBM25 protein, increased binding of RBM25 to SCN5A mRNA, increased SCN5A splice variant abundances, decreased full-length SCN5A mRNA and protein, and decreased Na + current."
22939879
reach
"Our results shows that AngII and hypoxia, signals common to HF, result in increased LUC7L3 and RBM25 splicing regulators, increased binding of RBM25 to SCN5A mRNA, increased SCN5A splice variant abundances, decreased full-length SCN5A mRNA and protein, and decreased Na + current."
22939879
reach
"In summary, AngII and hypoxia, signals common to HF, result in increased hLuc7A and RBM25 protein, increased binding of RBM25 to SCN5A mRNA, increased SCN5A splice variant abundances, decreased full-length SCN5A mRNA and protein, and decreased Na + current."
22939879
sparser
"Our results shows that AngII and hypoxia, signals common to HF, result in increased LUC7L3 and RBM25 splicing regulators, increased binding of RBM25 to SCN5A mRNA, increased SCN5A splice variant abundances, decreased full-length SCN5A mRNA and protein, and decreased Na + current."
22939879
reach
"Our results show that angiotensin II and hypoxia, signals common to HF, result in increased LUC7L3 and RBM25 splicing regulators, increased binding of RBM25 to SCN5A mRNA, increased SCN5A splice variant abundances, decreased full-length SCN5A mRNA and protein, and decreased Na (+) current."
22939879
reach
"RBM25 was bound to the wild-type SCN5A sequence in a concentration dependent manner (XREF_FIG)."
21859973