IndraLab

Statements

USP28 activates FOXC1. 3 / 3
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"In conclusion, USP28 enhanced cell viability and aerobic glycolysis of colorectal cancer by stabilizing FOXC1, suggesting that USP28-FOXC1 might be a novel therapeutic avenue for colorectal cancer."
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"Moreover, proteasome inhibitor, MG132, could rescue USP28 silence induced degradation of FOXC1."
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"Protein expression of Forkhead Box C1 (FOXC1) was increased by USP28 over-expression, whereas knockdown of USP28 aggravated cycloheximide (CHX; protein synthesis inhibitor) stimulated decrease of FOXC1."
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