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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

LMP1 activates TNFAIP3. 3 / 3

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"A20 is induced by LMP1 via NF-KappaB as a negative feedback mechanism to block further activation of both canonical and non-canonical NF-KappaB , and JNK activation [ 21 , 138 ] ."

34079586

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"A second possibility is A20, which is activated by both LMP1 ( Laherty et al. 1992 ) and CD40 ( Sarma et al. 1995 )."

9390690

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eidos

"As described in a previous section , A20 induction by EBV LMP1 was shown to inhibit IRF7 , which consequently prevents IRF7dependent IFN induction ( Ning and Pagano , 2010 ) ."

21847386

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...