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USP38 inhibits lactate. 3 / 3

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"Conversely, overexpression of USP38 inhibited PM2.5‐induced cell apoptosis (Figure 2D and Figure S1E) and reduced PM2.5‐induced lactate production (Figure 2F)."

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"In conclusion, these findings demonstrated that USP38 mitigates PM2.5‐induced lung epithelial cell damage by activating the AKT/mTOR pathway via MCT1, reducing lactate accumulation and apoptosis.4 Discussion."

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"We have demonstrated that both USP38 and MCT1 reduce PM2.5‐induced lactate accumulation."

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USP38 inhibits lactate. 3 / 3

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