IndraLab

Statements

Entinostat increases the amount of KCNH2. 4 / 4
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"Interestingly, KCNH2 and Cav1.3 protein expression levels could be partially restored by Entinostat in failing, stimulated hearts."
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"Entinostat treatment partially restored KCNH2 and Cav1.3 gene expressions in failing hearts, and inhibited the development of cardiac fibrosis in vivo."
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"KCNH2 and Cav1.3 were downregulated in response to pacing, and Entinostat was able to partly restore KCNH2 and Cav1.3 expression in failing hearts."
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"In a rabbit model of heart failure, Entinostat diminishes heart failure related prolongation of repolarization and partially restores KCNH2 and Cav1.3 expression."
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