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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP14 inhibits endoplasmic reticulum. 4 / 4

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"USP14 and UCHL5 inhibitors induce accumulation of ubiquitinated proteins and endoplasmic reticulum stress response in ER + BCa."

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"Notably, in resting cells, USP14 tonically blocks ER associated degradation by interaction with IRE1alpha."

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"Inhibition of USP14 induces ER stress mediated autophagy without apoptosis in lung cancer cell line A549."

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"Inhibition of USP14 and UCHL5 activates caspase and triggers apoptosis of ER + BCa cells."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

USP14 inhibits endoplasmic reticulum. 4 / 4

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