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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

BRCC3 activates homologous recombination. 2 / 2

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reach

"Although the suppressive role of BRCC36 in RNF8 dependent DDR suggests that BRCC36 promotes HR, the depletion of BRCC36 increases HR efficiency [XREF_BIBR]."

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reach

"Interestingly, the depletion of BRCC36 increases HR, which goes against the suppressive role of BRCC36 in RNF8 dependent DDR that should suggest that BRCC36 promotes HR."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...