IndraLab

Statements


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"Conversely, overexpression of USP38 accelerates the progression of cardiac hypertrophy and myocardial fibrosis."

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"Herein, we found that USP38 positively regulates pathological cardiac hypertrophy and remodeling by preventing the proteasomal degradation of p-TBK1, leading to the activation of Akt-GSK3β/mTOR signaling pathway and the acceleration of heart failure progression."