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USP18 activates LC3-II. 2 / 2

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"Consistent with the role of USP18 as a negative regulator of type I IFN signaling,6 knockdown of USP18 further potentiated the reduction of LC3-II induced by type I IFN."

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"Overexpression of USP18 led to upregulation of LC3-II and downregulation of SQSTM1 in a dose dependent manner."

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IndraLab

Statements

USP18 activates LC3-II. 2 / 2

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