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Mice activates USP38. 4 / 4
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"Conversely, Akt, GSK3β and mTOR phosphorylation levels were markedly increased in USP38 overexpression mice (Figure 5B)."
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"Next, we also established cardiac-specific USP38 overexpression mice to further verify the role of USP38 in HF induced by pressure overload (Fig. S3B)."
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"However, mice overexpressed USP38 had a higher incidence of VAs, with VT accounting for 75% and VF accounting for 20% (Fig. 3H, I)."
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"We constructed and generated cardiac specific USP38 knockout mice and cardiac specific USP38 overexpression mice to assess the role of USP38 in pathological cardiac remodeling."
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