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CACNA1B binds HTT. 1 / 1

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"The association of the N-terminal domain of huntingtin (both mutant and normal) with the pore-forming CaV2.2 subunit of N-type VGCC leads to a displacement of the syntaxin 1A that negatively regulates the channel and, as a result, to an increase in the activity of N-type VGCC [ xref ]."

28740725

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IndraLab

Statements

CACNA1B binds HTT. 1 / 1

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