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VEGF activates CALM1. 15 / 15
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"(a) VEGF 0 (DMEM soaked implant), VEGF 30 (30 ng/implant), VEGF 100 (100 ng/implant) and VEGF 300 (300 ng / implant) induced CAM angiogenic response in absence of coronary endothelial cells (AcEC) and in presence of coronary endothelial cells (2 A 106 cells/ml, 0.5 A 104 cells and implant) of normal (+ ncEC) rats, diabetic rats 30 ds."

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"Effect of eNOS inhibitor, L-NAME (100 mM, l-N), PI3K inhibitor, wortmanin (100 nM, w) and PKC inhibitor, chelerythrine (10 mM, ch) pretreatment on (b) VEGF 100 induced CAM angiogenic response and (c) VEGF 300 (300 ng/ml) induced nitric oxide release activity of + TnircEC."

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"(a) VEGF 0 (DMEM soaked implant), VEGF 30 (30 ng/implant), VEGF 100 (100 ng/implant) and VEGF 300 (300 ng / implant) induced CAM angiogenic response in absence of coronary endothelial cells (AcEC), in presence of ischemia reperfusion injured coronary endothelial cells (2 A 106 cells/ml, 0.5 A 104 cells and implant) of diabetic 60 ds."

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"Effect of eNOS inhibitor, L-NAME (100 mM, l-N), PI3K inhibitor, wortmanin (100 nM, w) and PKC inhibitor, chelerythrine (10 mM, ch) pretreatment on (b) VEGF 100 induced CAM angiogenic response and (c) VEGF 300 (300 ng/ml) induced nitric oxide release activity of +30 / TdcEC."

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"Effect of eNOS inhibitor, L-NAME (100 mM, l-N), PI3K inhibitor, wortmanin (100 nM, w) and PKC inhibitor, chelerythrine (10 mM, ch) pretreatment on (b) VEGF 300 induced CAM angiogenic response and (c) VEGF 300 (300 ng/ml) induced nitric oxide release activity of +60 / TdcEC."

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"Effect of eNOS inhibitor, L-NAME (100 mM, l-N), PI3K inhibitor, wortmanin (100 nM, w) and PKC inhibitor, chelerythrine (10 mM, ch) pretreatment on (b) VEGF 100 induced CAM angiogenic response and (c) VEGF 300 induced nitric oxide release activity of + ncEC."

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"Effect of eNOS inhibitor, L-NAME (100 mM, l-N), PI3K inhibitor, wortmanin (100 nM, w) and PKC inhibitor, chelerythrine (10 mM, ch) pretreatment on (b) VEGF 100 induced CAM angiogenic response and (c) VEGF 300 (300 ng/ml) induced nitric oxide release activity of + TncEC."

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"However, it is possible that VEGF was not present in sufficient concentration to stimulate the CAM in Fuchs et al 's experiments (1985) and that the placental tissue contained other growth factors wit[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"VEGF stimulates NO production initially by increasing intracellular Ca 2 + levels and subsequent Ca 2 + -calmodulin dependent activation of eNOS, and later by increasing intracellular eNOS message and[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"Contributions to endothelial activation and proangiogenic processes are made by CAM-1 and ICAM-1, which can be induced by vascular endothelial growth factor (VEGF) and cytokines [65]."

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"(a) VEGF 0 (DMEM soaked implant), VEGF 30 (30 ng/implant), VEGF 100 (100 ng/implant) and VEGF 300 (300 ng / implant) induced CAM angiogenic response in absence of coronary endothelial cells (AcEC), in presence of ischemia reperfusion injured coronary endothelial cells (2 A 106 cells/ml, 0.5 A 104 cells and implant) of diabetic 30 ds."

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"Endostatin-XV did not reduce endothelial cell sprouting from bone explants although endostatin-XV had been shown to reduce VEGF stimulated angiogenesis in CAM assays (Sasaki et al., 2000)."

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"(a) VEGF 0 (DMEM soaked implant), VEGF 30 (30 ng/implant), VEGF 100 (100 ng/implant) and VEGF 300 (300 ng / implant) induced CAM angiogenic response in absence of coronary endothelial cells (AcEC), in presence of coronary endothelial cells (2 A 106 cells/ml, 0.5 A 104 cells / implant) of normal (+ ncEC) rats and telmisartan (2 mg/kg, p.o., 15 days, + TncEC) treated normal rats on 4th day after implantation."

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"VEGF 0 (DMEM soaked implant), VEGF 30 (30 ng/implant), VEGF 100 (100 ng/implant) and VEGF 300 (300 ng/implant) induced CAM angiogenic response in absence of coronary endothelial cells (AcEC) and in presence of ischemia reperfusion injured coronary endothelial cells (0.5 A 104 cells and implant) of normal (+ nircEC) rats, diabetic rats 30 ds."

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"CPRECESIC peptide (1 mM) inhibited VEGF induced CAM neovascularization by 40% (35.5 +/- 1.4 blood vessels; t test, p < 0.01)."