IndraLab

Statements


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"Overall, USP38 overexpression aggravates pressure overload-induced pathological cardiac remodeling and promotes the development of heart failure."

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"In this study, we examined the impact of USP38 on VAs in pressure overload-induced HF."

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"These results suggest that USP38 deficiency alleviates cardiac hypertrophy and myocardial fibrosis, and prevents the progression of heart failure."