IndraLab

Statements

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"USP39 knockdown inhibits clonogenic capacity, induces cell cycle arrest, triggers apoptosis, and overcomes BTZ resistance."
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"Knockdown of USP39 can promote apoptosis and cell cycle arrest in HCC cells, and SP1 reverses these effects (26)."
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"Our previous studies have established that USP39 knockdown induces cell cycle arrest at the G2/M phase in non-small cell lung cancer (NSCLC) and colorectal cancer cells, subsequently triggering apoptosis through activation of the p53 pathway [40, 41]."
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