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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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CYLD inhibits cell adhesion. 3 / 3

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"These findings suggest that age related attenuation of CYLD expression in endothelial cells (ECs) and macrophages triggers the initiation of age related atherogenesis by exacerbating monocyte adhesion on the endothelium and foam cell formation."

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"siRNA mediated CYLD silencing led to enhanced monocyte adhesion along with increased adhesion molecules in HAECs treated with TNFalpha."

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"Furthermore, adhesion of fluorescein isothiocyanate labeled THP-1 cells to BAECs was also inhibited by CYLD overexpression."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

CYLD inhibits cell adhesion. 3 / 3

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