IndraLab

Statements

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"Similarly, the deletion of USP44, an integral component of NCoR, impaired the ability of NCoR to regulate gene expression and suppressed breast cancer cell invasiveness [29]."
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"USP44 inhibits proliferation, self-renewal, migration, and invasion of OSCC cells in vitro."
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"Downregulation of USP44 expression can inhibit proliferation, migration and invasion of established glioma cell lines and induce apoptosis (85)."
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"Furthermore, we found that knockdown of USP44 in CAL-27 and SCC-9 cells noticeably promoted cell migration and invasion, whereas its overexpression substantially impeded cell migration and invasion (Fig. 2E and H)."
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"As a result, USP44 overexpression suppressed cellular invasion and migration, while downregulation of Itch alleviated the inhibition of cellular invasion and migration induced by USP44 overexpression (Fig. 5D, E)."
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"Knockdown of USP44 inhibited proliferation, migration and invasion, induced apoptosis, and arrested cell cycle in G2/M phase in the established glioma cell lines."
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"At the same time, CCK-8 and Transwell assays also revealed that while overexpression of USP44 (pLJM1-USP44) inhibited the proliferation, migration, and invasion of CAL-27 cells, these effects were notably reversed upon HEXIM1 knockdown (Fig. 8D and E)."
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"Depletion of USP44 inhibits proliferation, migration, and invasion in glioma cell lines."
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"Functional assays showed that increased USP44 expression notably suppressed OSCC cell proliferation, migration, invasion and cancer stem cell-like behaviors, whereas its decreased expression had the opposite effects."
39722007
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"Depletion of USP44 significantly impaired the invasiveness of MDA-MB-231 cells in vitro and led to an increase of global H2Bub1 levels (XREF_FIG and XREF_SUPPLEMENTARY)."
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