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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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SCN9A activates sodium atom. 3 / 3

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"Recently, SCN9A gene variants (single amino acid substitutions) have been found in ~ 30% of a cohort of idiopathic SFN patients, producing gain-of-function changes in sodium channel Na (V) 1.7, which is preferentially expressed in small diameter peripheral axons."

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"Knockout of Scn9a lowers Na + current by> 85%, disclosing a small Scn3a dependent component."

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"In beta-cells, knockout of Scn9a lowers the Na + current by> 85%, unveiling a small Scn3a dependent component."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

SCN9A activates sodium atom. 3 / 3

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