IndraLab

Statements

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"Furthermore , we found that CYLD deficiency promoted cervical cancer cell proliferation , colony formation , cell migration and invasion , and inhibited apoptosis instead , whereas it is just opposite with CYLD over-expression ."
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"Histologically, CYLD clearly prevented massive immune cell infiltration surrounding necrotic regions, and pseudopalisades appeared in bevacizumab treated control tumors."
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"For example, inhibition of CYLD expression enhanced human malignant melanoma growth and invasion [XREF_BIBR]."
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"XREF_BIBR Additionally, Sanches et al XREF_BIBR showed that CYLD suppresses cell migration and invasion in cervical cancer, and Suenaga et al XREF_BIBR reported that CYLD downregulates induction of cisplatin resistance in oral squamous cell carcinoma."
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"Together, these findings suggest that CYLD restricts infiltration of inflammatory cells and invasion enhanced by increased hypoxia after anti-VEGF therapy in GBM."
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"Loss of CYLD stimulates cellular proliferation, migration, and invasion by triggering BCL-3 nucleus translocation and activation of cyclin D1 and N-cadherin [XREF_BIBR]."
| PMC
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"Loss of CYLD stimulates cellular proliferation , migration , and invasion by triggering BCL-3 nucleus translocation and activation of cyclin D1 and N-cadherin [ 99 ] ."
| PMC
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"Studies found that miR-362-5p reduces the expression of tumor suppressor protein CYLD, which leads to activate the NF-kB pathway to promote the proliferation, migration, and invasion of hepatocellular carcinoma cells and human breast cancer cells (Ni et al., 2015; Ni et al., 2016)."
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"Furthermore, CYLD overexpression inhibited SMAD7 mediated cell invasion, while CYLD depletion increased SMAD7 mediated cell invasion in OSCC cells."
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"These clinical findings support a link between the known tumor promoter Snail1 and its transcriptional target CYLD, indicating an important role for Snail1 to suppress CYLD during progression of melanoma and for CYLD to suppress melanoma cell proliferation and invasion."
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"Rescue of CYLD expression in melanoma cells reduced proliferation and invasion in vitro and tumor growth and metastasis in vivo."
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"Compared with the control group, miR-181 silencing or CYLD overexpression significantly attenuated cell proliferation, invasion and migration, and notably increased the proportion of apoptotic cells."
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"Loss of CYLD promotes cell invasion via ALK5 stabilization in oral squamous cell carcinoma."
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"Treatment with sorafenib, EAC and sorafenib/EAC inhibited cell invasion by 57.0%, 67.5% and 86.4%, respectively (Fig.  xref )."
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