IndraLab

Statements


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"In the present study we also have demonstrated that the nitric oxide modification which is likely to happen in nitrosative stress or excitotoxicity condition destabilizes the UCHL1 structure and induces the aggregation of UCHL1."

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"Collectively, above findings indicated that increase of cellular nitric oxide promoted UCHL1 nitrosylation of three critical cysteine residues (XREF_FIG)."

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"Rotenone treated SH-SY5Y cells enhance NO production thereby promoting S nitrosylation of UCHL1."