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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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CYLD activates NFKB1. 3 / 4

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"CYLD activates NFKB signaling through deubiquitination of NFKB upstream regulators ubiquitylated by cIAP ubiquitin E3 ligase, and inhibition of cIAP by cIAP antagonist blocks NFKB activation XREF_BIBR XREF_BIBR."

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"Cyld inhibits tumor cell proliferation by blocking Bcl3 dependent NFkB signaling."

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"The para-caspase mucosa associated lymphoid tissue (MALT-1) has recently emerged as the most critical regulator of CYLD- mediated NFkB activation in various immune and non immune cells [6-8]."

25130432

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Statements

CYLD activates NFKB1. 3 / 4

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