IndraLab

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"In addition, cAMP was supposed to negatively modulate the interaction of TREK-1 with POPDC proteins."
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"Interestingly, cAMP analogs have been reported to enhance TREK1 mRNA independently from PKA in adrenocortical cells XREF_BIBR."
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"Interestingly, as outlined in Table 5, a vast majority of identified binding and reaction partners of AKAP5 is altered in BD, being either up-regulated (cAMP dependent protein kinase A and its regulat[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"This suggested that the loss of cAMP-controlled modulation of TREK1 currents was in part responsible for the cardiac arrhythmias observed in patients possessing the POPDC1 mutation and corresponding animal models [14]."
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