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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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N-acetyl-beta-D-galactosaminyl-(1->4)-N-acetyl-D-glucosaminyl group inhibits UCHL1. 4 / 4

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"In this study, we examined the effect of UCHL1 inhibition by LDN, a specific inhibitor, on the development of AF in a murine model of Ang II-induced AF."

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"Both inhibition of UCHL1 by LDN and knockdown UCHL1 by siRNA downregulated TGF-β1-mediated expression of pro-fibrotic proteins in CFs."

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"To assess whether UCHL1 mediates post-MI cardiac fibrosis in vivo, we used LDN to inhibit UCHL1."

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"The inhibition of UCHL1 by LDN (a specific inhibitor) significantly reduced the Ang II-induced increase in blood pressure, atrial fibrillation, fibrosis, inflammation, and oxidative stress."

31700166

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Statements

N-acetyl-beta-D-galactosaminyl-(1->4)-N-acetyl-D-glucosaminyl group inhibits UCHL1. 4 / 4

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