IndraLab

Statements



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"Therefore, we assessed whether USP18 could modulate the EMT phenotype in ESCC cells."

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"Furthermore, our results shown that knockdown of USP18 suppressed the migration and invasion abilities of ESCC cells by promoting the process of epithelial-mesenchymal transition (EMT)."

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"USP18 could interact with Twist1, and increased the activity of EMT pathway, which promoted glioma growth [18]."