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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

VCPIP1 inhibits Top1cc. 2 / 2

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"Loss of VCPIP1 resulted in cellular hypersensitivity to DPC-inducing agents, as well as Top1cc and DPC accumulation, suggesting that VCPIP1 plays a critical role in DPC repair."

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"We found that VCPIP1 deficiency resulted in enhanced Top1cc foci signals after CPT treatment (Figures 1M–1N), suggesting a role of VCPIP1 in Top1cc removal."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...