IndraLab

Statements

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"Further RT-qPCR experiments showed that LPS stimulation induced significant downregulation of miR-206 (Fig. 1B) and remarkable upregulation of USP33 (Fig. 1C)."
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"In addition, the activation of JAK2/STAT3 by coumermycin A1 remarkably reversed the protective role of miR-206 in LPS-induced inflammatory injuries, suggesting JAK2/STAT3 signaling was a key downstream pathway of miR-206/USP33 in SCM.In this study, our data revealed that miR-206 alleviated LPS-induced inflammatory injuries by directly targeting USP33 and suppressing the activation of JAK2/STAT3 signaling pathway."
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"Correction: miR-206 alleviates LPS-induced inflammatory injury in cardiomyocytes via directly targeting USP33 to inhibit the JAK2/STAT3 signaling pathway."
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"miR-206 alleviates LPS-induced inflammatory injury in cardiomyocytes via directly targeting USP33 to inhibit the JAK2/STAT3 signaling pathway."
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"Gain-of-function of miR-206 promoted the proliferation but suppressed the inflammatory responses and apoptosis of LPS-induced cardiomyocytes by targeting USP33 and suppressing the JAK2/STAT3 signaling pathway."
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