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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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Statements

CYLD-C601A activates NFKBIA. 1 / 1

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"Consistent with our previous study [XREF_BIBR], the inhibitory function of CYLD on NF-kappaB relies on its intact catalytic activity because expression of a catalytically inactive CYLD C601A mutant, in which cysteine601 is substituted by alanine, could not enhance the abundance of IkappaBalpha to the equivalent extent as wild-type CYLD did (XREF_SUPPLEMENTARY)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...