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"Results of flow cytometry then showed that UCHL3 knockdown augmented cell apoptosis, along with increased cells arrested in the G0/G1 phase, decreased cells arrested in the S phase, and repressed cell division, while the restoration of UCHL3 caused opposite results (Fig. 3E, F)."
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"Programmed cell death is implicated in a number of human diseases, including neurodegenerative disease, 3 autoimmune disease, 4 cancers 5,6 , etc. Loss of UCH-L3 leads to programmed cell death by apop[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Furthermore, under conditions of IR, the level of apoptosis increased by silencing UCHL3 was overcome by POLD4 overexpression in MES GSCs (Fig. 6D and Fig. S7A)."
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