IndraLab

Statements

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"These results suggested that knockdown of USP44 might inhibit glioma cell proliferation by arresting cell cycle in G2/M phase."
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"We observed that depletion of USP44 slows proliferation in a dose-dependent fashion whereas overexpression promotes it (Fig. 3A and D)."
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"USP44 accelerated the proliferation and cell cycle progression of T-ALL cells."
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"Overexpression of USP44 promoted the proliferation of CCRF-CEM cells."
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"However, the knockdown of USP44 impeded the proliferation of Jurkat and MOLT-4 cells (Figure 2A)."
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"These results indicated that USP44 could promote the proliferation and accelerate the cell cycle progression of T-ALL cells."
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"Overall, the difference derived from our experimental results indicated that the knockdown of USP44 inhibited proliferation, migration, and invasion in glioma cells."
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"XREF_BIBR The downregulation of miR-143-3p by circFoxo3 increased the level of USP44, which in turn promoted tumor cell proliferation."
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"Indeed, knockdown of cyclin D1 or re-expression of USP44 reversed the proliferation of T80 and KLF8 cells back to the levels of the Mock cells (XREF_FIG)."
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"In established glioma cell lines, knockdown of USP44 inhibited the proliferation, migration, and invasion of the cells."
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"It was also demonstrated that downregulation of USP44 promoted cell proliferation and migration through the JNK pathway in renal clear cancer cells."
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"The results from CCK-8 and EdU assays showed that the upregulation of USP44 significantly inhibited cell growth, while Itch knockdown impaired the decline in cell proliferation induced by USP44 overexpression (Fig. 5A–C)."
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"In contrast, the knockdown of USP44 enhanced cell growth, whereas Itch overexpression mitigated the increase in cell proliferation enhanced by USP44 knockdown (Supplementary Fig. 4A–C)."
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"USP44 hypermethylation promotes cell proliferation and metastasis in breast cancer."
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"In our study, USP44 was highly expressed in T-ALL patients and accelerated the proliferation of T-ALL cells, indicating that USP44 could become a candidate target for treating T-ALL."
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"USP44 overexpression elevated the proliferation of CCRF-CEM cells, while USP44 knockdown suppressed the proliferation of Jurkat and MOLT-4 cells."
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"These results indicated that knockdown of USP44 inhibited the proliferation of glioma cells."
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