IndraLab

Statements


CACNA1C increases the amount of ATP. 2 / 2
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"In line with the previous findings on mitochondrial morphology, the downregulation of Cacna1c blocked the loss of ATP levels in response to glutamate."

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"Nevertheless, Cav1.2 channel signaling through endoplasmic reticulum ryanodine receptors stimulated mitochondrial OXPHOS to help maintain cytosolic adenosine triphosphate (ATP) levels necessary for pacemaking."