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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP29 activates Reperfusion Injury. 6 / 6

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"Collectively, these findings imply that USP29 potentiates I/R-induced oxidative damage and neuronal apoptosis in vitro."

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"USP29 Potentiates I/R-Induced Oxidative Damage and Neuronal Apoptosis In Vitro."

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"USP29 Inhibition Prevents Cerebral I/R Injury in Mice."

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"The findings enable us to believe that USP29 inhibition prevents cerebral I/R injury in mice."

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"USP29 Inhibition Diminishes Cerebral I/R-Induced Oxidative Stress and Apoptosis."

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"Our previous study identified a protective effect of SIRT3 against cerebral I/R injury; therefore, we investigated whether USP29 inhibition prevented cerebral I/R injury through upregulating SIRT3 [28]."

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USP29 activates Reperfusion Injury. 6 / 6

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