IndraLab

Statements

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"High doses of sodium salicylate suppressed NF-kappa B p65 mRNA accumulation, resulting in suppression of total NF-kappa B, p50 on tissue oligonucleotide had no effects on lipopolysaccharide induced NF-kappa B activation."
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"The data demonstrate that the p65 subunit of NF-kappa B is inhibited by salicylate treatment and highlight the role of salicylate in the control of gene expression of inflammatory mediators."
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"Overall, our data suggest that the activation of PPAR-γ signaling attenuates NF-κB and PAK1 signaling in vivo and in vitro, thereby inhibiting the degranulation of mast cells.Previous studies have sho[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Inhibition of nuclear factor kappa B subunit p65 mRNA accumulation in lipopolysaccharide stimulated human monocytic cells treated with sodium salicylate."
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