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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

CYLD activates RELA. 2 / 2

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"Song et al. showed that miR-301b was upregulated in TNBC specimens and cell lines, and inhibited the apoptosis induced by 5-FU by direct binding to 3′-UTR of CYLD lysine 63 deubiquitinase (CYLD) mRNA to activate NF-κB p65 [169]."

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"Thus, Romidepsin NF-kappaB p65 downregulation may be mediated, at least in part, by CYLD overexpression."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...