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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

UCHL3 activates INS. 2 / 2

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"Suzuki et al. have shown that UCHL3 enhances insulin signalling in (pre) adipocytes, but UCHL3 is unlikely to target critical components of insulin signalling like the insulin receptor, the IGF-I receptor, IRS-1 and Grb10 XREF_BIBR."

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"These results suggest that UCH-L3 promotes adipogenesis by enhancing insulin signaling in a hydrolase activity dependent manner."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...