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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

UCHL1 activates ACTN4. 1 / 1

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"Our data suggest that UCHL1 upregulation in ACTN4 associated FSGS fuels the proteasome and that UCHL1 deletion may impair proteolysis and thereby preserve K256E and wt-alpha-actinin -4 heterodimers, maintaining podocyte cytoskeletal integrity and protecting the glomerular filtration barrier."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...