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FSTL1 activates USP10. 3 / 3
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"The present study revealed that FSTL1 and USP10 were significantly activated in T2DM mice with MI, and FSTL1 treatment further increased USP10 activation and alleviating cardiac dysfunction by reducing myocardial fibrosis."
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"Further in vitro analysis provided evidence that FSTL1 activated USP10/Notch1 signaling to inhibit myocardial fibrosis."
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"Follistatin-like protein 1 (FSTL1) has been shown to activate the USP10/Notch1 signaling pathway, which is associated with myocardial apoptosis in T2DM mice."
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