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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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FOXO4 decreases the amount of USP10. 3 / 3

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"FoxO4 overexpression could reverse the protective effects of USP10 overexpression on H/R induced H9C2 cells by regulating the Hippo and YAP signaling pathway."

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"FoxO4 negatively modulates USP10 transcription to aggravate the apoptosis and oxidative stress of hypoxia and reoxygenation induced cardiomyocytes by regulating the Hippo and YAP pathway."

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"FoxO4 negatively modulates USP10 transcription to aggravate the apoptosis and oxidative stress of hypoxia/reoxygenation-induced cardiomyocytes by regulating the Hippo/YAP pathway."

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Statements

FOXO4 decreases the amount of USP10. 3 / 3

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