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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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BAP1 increases the amount of FGFBP1. 3 / 3

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"WT BAP1, but not mutant BAP1 C91S, obviously elevated the KLF5 and FGF-BP protein levels and inhibited the p27 protein levels in both cell lines (XREF_FIG)."

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"As expected, knockdown of BAP1, HCF-1, OGT or KLF5 markedly increased p27 and decreased FGF-BP protein levels in HCC1806 cells (XREF_FIG)."

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"In addition, expression of KLF5, CyclinD1, and FGF-BP1 was increased by BAP1 overexpression and decreased by BAP1 knockdown."

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Statements

BAP1 increases the amount of FGFBP1. 3 / 3

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