IndraLab

Statements



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"The proliferation of colorectal cancer cells is suppressed by silencing of EIF3H."

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"Our results suggest that eIF3h content is near a threshold in Hap1 cells, since overexpression of SNAP-eIF3H appears to diminish wild-type eIF3h protein levels and fails to increase the proliferation rate of wild-type cells."

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"EIF3h potentiates the cell proliferation and inhibits the cell apoptosis of gastric cancer cells 13."

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"Knockdown of EIF3H inhibited cell proliferation and colony formation ."

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"Previous studies have shown that RFFL and HDAC2 are known to also promote tumor formation by inhibiting the apoptosis process [XREF_BIBR, XREF_BIBR, XREF_BIBR], whereas EIF3H is known to increase cell proliferation, growth and survival and inhibit the apoptosis process [XREF_BIBR]."

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"EIF3H overexpression increases tumor cell proliferation, growth, and survival."

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"Finally, reduction of eIF3h levels in breast and prostate cancer cell lines by short interfering RNA methods reduces their rates of proliferation and anchorage independent growth in soft agar."

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"The higher polyp number associated with the C allele of rs16892766 could be explained by the location of this SNP in the EIF3H gene, which increases cell proliferation, growth, and survival when overexpressed."

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"Reduction of eIF3h levels in breast and prostate cancer cell lines by shRNA methods has previously been shown to reduce cell proliferation and anchorage independent growth in soft agar XREF_BIBR."

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"XREF_BIBR At the cellular level, EIF3H overexpression increases proliferation, growth and survival."

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"Reduction of EIF3H has previously been shown to reduce cell proliferation and anchorage independent growth in soft agar [XREF_BIBR]."

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"We observed that EIF3H overexpression in both KYSE150 and KYSE510 cells increased cell proliferation in vitro ( Fig. 2e and f ) , whereas depletion of EIF3H significantly reduced cell proliferation ( Fig. 2g and h ) ."

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"In the LoVo CRC line, reduction of EIF3H levels by short interfering RNA (shRNA) reduced cell proliferation (XREF_FIG)."

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"At the cellular level, EIF3H overexpression increases proliferation, growth and survival."

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"As shown in XREF_FIG, knockdown of endogenous EIF3H significantly inhibited the proliferation of Saos-2 and U2OS cells at 24, 48, 72, 96 and 120 h after viral infection."

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"Knockdown of EIF3H inhibits the development and progression of pancreatic cancer by regulating cell proliferation and apoptosis in vitro."

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"We observed that EIF3H overexpression in both KYSE150 and KYSE510 cells increased cell proliferation in vitro, whereas depletion of EIF3H significantly reduced cell proliferation."

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"Conversely, EIF3H up-regulation by transfection with lentivirus carrying an EIF3H expression vector (pWP1-EIF3H) increased cellular proliferation (XREF_FIG)."

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"Thus, we come to the conclusion that EIF3H promotes proliferation and migration of iCCAs, inhibiting apoptosis of iCCA cells at the same time by stabilizing the CCND1 protein structure."