IndraLab

Statements

METTL3 activates USP12. 4 / 4
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"This result suggested that METTL3 may mediate the pro-hypertrophic function of USP12."
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"METTL3 acted as a pathogenic factor and mediated the pro-hypertrophy effect of USP12 in Ang-II-induced myocardium remodeling [9]."
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"These data led us to investigate whether METTL3 mediated the pro-hypertrophic function of USP12."
34339675
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"Importantly, upregulation of METTL3 reversed the decrease of Ang II-induced myocardial hypertrophy in USP12-silenced NRCMs."
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