IndraLab

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"In this study, overexpression of USP18 further raised the levels of inflammatory factors, and promoted the synthesis and secretion of IFN-β and chemokine CXCL10 in IAV-infected A549 cells."
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"However, according to research by Hong et al., increased USP18 expression in tumor cells would in turn inhibit carcinogenesis, whereas decreased USP18 promotes tumor growth and lowers immunosurveillance by decreasing the exogenous synthesis of IFN-γ and the survival of cytotoxic T lymphocytes (CTLs) in TME [178]."
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"What’s more, overexpression of USP18 further enhanced the synthesis and secretion of IL-6, TNF-α, IFN-β and CXCL10 after PR8 infection ( Fig. 2 A and B), while inhibition of USP18 had an opposite effe[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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