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"We infer that inhibition of BPDE-induced apoptosis by cadmium may represent a possible mechanism for its co-carcinogenic function in presence of BPDE as DNA damaging agent.Our results also show that i[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"When cells were treated with ASO c-Jun, the total amounts of c-Jun and also cadmium induced c-Jun activation were diminished."

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"Enhanced expression of p53 and apoptosis in ventral prostate (not the case in testes) may be a tissue reaction working to diminish the activation of c-jun by cadmium."