IndraLab

Statements

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"These observations suggested that the proinflammatory DC phenotype was induced by the caerin gel.Caerin gel treatment significantly upregulated the expression of Tnfaip3 in NK cells."
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"20 , 21 Several models of DC‐specific deletion of TNFAIP3 in CD11c cells have shown that this molecule limits DC activation, and in the absence of TNFAIP3, DCs produce higher levels of cytokines, express higher co‐stimulatory molecules, resist to cell death, and activate auto‐reactive T‐ and B‐cell responses."
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